- ￥6600
- 赛默飞（Thermo Fisher）
- PA5110946
- 2025年07月12日
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文献和实验Ier1 p: A Kinase and Site-Specific Endoribonuclease
fibrosis, alpha1-antitrypsin deficiency, and osteogenesis imperfecta (2 ). One way in which cells cope with the accumulation of unfolded proteins in the ER is by activating the unfolded protein response (UPR), an ER-to-nucleus signal transduction pathway
hydroxylaseAssociation of a 14-3-3 protein with CMP-NeuAc:GM1 alpha 2,3-sialyltransferaseInteraction of 14-3-3 with signaling proteins is mediated by phosphoserineActivation-modulated association of 14-3-3 proteins with Cbl14-3-3 binding sequence in cytoplasmic domain
Introduction of human 14-3-3 proteins
terminus of GPIb-alpha,as indicated by antibody inhibition and direct binding of 14-3-3zeta to synthetic GPIb-alpha cytoplasmic domain peptides.The 14-3-3zeta binds to recombinant wild type GPIb-IX but not to the GPIb-alpha mutants lacking C-terminal
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