- ￥5707
- 赛默飞（Thermo Fisher）
- MA549316
- 2025年07月11日
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文献和实验at 10, 25, and 50 μM). At 24 h post-treatment, celllysates were obtained; SDS-PAGE and Western blotting were performed as described in Section 3, Methods. AR-A014418dose-dependent decreases of phospho-β-catenin (Ser33/37) and phospho-glycogen synthase
domains as well as ataxia telangiectasia mutated (ATM) protein kinase in cells treated with inhibitors of DNA replication. Phosphorylation of these proteins is detected in individual cell immunocytochemically with phospho-specific antibody (Ab
Cytometric Assessment of DNA Damage Induced by DNA Topoisomerase Inhibitors
by phosphorylation of histone H2AX on Ser- 139 which is mediated by one of the protein kinases of the phosphoinositide kinase family, namely ATM, ATR, and/or DNA-PK. The presence of Ser -139 phosphorylated H2AX (γH2AX) is thus a reporter of DNA damage. This protocol
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