Recombinant-Glycine-max-Chloro
plast-envelope-membrane-proteincemAChloroplast envelope membrane protein- ￥10822
- CUSABIO
- CN
- CSB-CF343777GGV
- 2025年07月15日
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- 文献和实验
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- 保存条件：
"-20°C/-80°C"
- 保质期：
Generally, the shelf life of liquid form is 6 months at -20°C/-80°C. The shelf life of lyophilized form is 12 months at -20°C/-80°C.
- 英文名：
Customized Glycine max cemA Protein (in vitro E.coli)
- 库存：
200
- 供应商：
武汉华美生物工程有限公司
- 规格：
20ug
Alternative Name(s)
Chloroplast envelope membrane proteinEditorial/Sponsord
EditorialUniprot ID
P49160Gene Names
cemAOrganism
Glycine maxAASequence
MTKKKTFIPLLYLTSIVFLPWCISFTFKKSLESWFIDWWNTRQSEIFLNDIKEKSILKKF IEFEELFFLDDMLKECPETHLQNLRTGIYKETIQLIKTHNEDRMNTILHFSTNIICFFIL SGYSILGNQELVLINSLVREFIYNLSDTIKAFSILLLTDLCIGFHSTHGWELVIGFVYKD FGFAQNDQIISGLVSTFPVILDTILKYWIFRYLNRVSPSLVVIYHSMNDExpression Region
1-229aaSequence Info
Full lengthSource
in vitro E.coliSource Notice
Mammalian cell expression systems and other species are available. Please inquire.Tag Info
InquireMW
InquireList Price
1546Purity
Greater than 85% as determined by SDS-PAGE.Storage Buffer
Tris/PBS-based buffer, 6% TrehaloseStorage
The shelf life is related to many factors, storage state, buffer ingredients, storage temperature and the stability of the protein itself. Generally, the shelf life of liquid form is 6 months at -20°C/-80°C. The shelf life of lyophilized form is 12 months at -20°C/-80°C.Endotoxin
Not Test. Endotoxin removal service is available for free upon you request.产品类型
Transmembrane-Protein备注
**产品信息可能有变动，请以官网信息为准
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文献和实验**产品信息可能有变动，请以官网信息为准
9(3):301–309, 2008). Integrin α4 β7 is not an entry receptor for HIV-1, although it can facilitate virion attachment to target cells (Arthos et al., Nat Immunol 9(3):301–309, 2008; Cicala et al., Proc Natl Acad Sci U S A 106:20877–20882, 2009). Recombinant
A Cell-Cell Fusion Assay to Monitor HIV-1 Env Interactions with Chemokine Receptors
, after which the viral Env protein must be triggered to undergo a conformational change that mediates membrane fusion. Cell-surface binding has long been known to occur via a high-affinity interaction between Env and CD4. However, the cell-surface molecules responsible
选择性转移治疗基因至靶细胞 通过修饰或改造基因传递载体，赋予载体与靶组织或靶器官选择性结合的能力，从而达到将治疗基因选择性转移到靶组织或靶器官的目的。如前所述，反转录病毒载体在感染靶细胞时，需要其外壳蛋白(Env)与细胞表面的受体相互识别，因此可以通过修饰env基因或Env蛋白来改变其感染能力或嗜性。例如采用能识别肿瘤细胞表面受体的配体或针对肿瘤特异性抗原的抗体基因取代env基因部分序列，使病毒表达能特异性识别肿瘤细胞的外壳蛋白，然后将病毒选择性带人靶组织或靶细胞
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