Recombinant-Mitochondrial-fiss
ion-process-protein-1mtp-18Mitochondrial fission process protein 1 Alternative name(s): Mitochondrial 18 kDa protein; MTP18- ￥10220
- CUSABIO
- CN
- CSB-CF474147CXX
- 2025年07月08日
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- 详细信息
- 文献和实验
- 技术资料
- 保存条件：
"-20°C/-80°C"
- 保质期：
Generally, the shelf life of liquid form is 6 months at -20°C/-80°C. The shelf life of lyophilized form is 12 months at -20°C/-80°C.
- 英文名：
Customized Caenorhabditis briggsae CBG28141 Protein (in vitro E.coli)
- 库存：
200
- 供应商：
武汉华美生物工程有限公司
- 规格：
20ug
Alternative Name(s)
Mitochondrial fission process protein 1 Alternative name(s): Mitochondrial 18 kDa protein; MTP18Editorial/Sponsord
EditorialUniprot ID
B6IJ52Gene Names
CBG28141Organism
Caenorhabditis briggsaeAASequence
MSPVESSNVEKDIFRDTPVRFLGKRYANEVGEAFRSLVKPVVVKFSYVVAFGYVAADSVD KGFKESKKPHANDTEKAKRVAIIAVDTVLWQTFASVLIPGFTINRFCFFTNMLLEKSTKL PTNLRKWTVTALGLATIPFIVHPIDAFVEDAMNKTARKVYNYExpression Region
1-162aaSequence Info
Full lengthSource
in vitro E.coliSource Notice
Mammalian cell expression systems and other species are available. Please inquire.Tag Info
InquireMW
InquireList Price
1460Purity
Greater than 85% as determined by SDS-PAGE.Storage Buffer
Tris/PBS-based buffer, 6% TrehaloseStorage
The shelf life is related to many factors, storage state, buffer ingredients, storage temperature and the stability of the protein itself. Generally, the shelf life of liquid form is 6 months at -20°C/-80°C. The shelf life of lyophilized form is 12 months at -20°C/-80°C.Endotoxin
Not Test. Endotoxin removal service is available for free upon you request.产品类型
Transmembrane-Protein备注
**产品信息可能有变动，请以官网信息为准
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文献和实验**产品信息可能有变动，请以官网信息为准
In Vivo Labeling and Analysis of Mitochondrial Translation Products in Budding and in Fission Yeasts
Mitochondrial biogenesis requires the contribution of two genomes and of two compartmentalized protein synthesis systems (nuclear and mitochondrial). Mitochondrial protein synthesis is unique on many respects, including the use of a genetic code
to Bax, a pro-apoptotic protein. However, the mechanistic implications of interactions between the machinery of mitochondrial fission and fusion and apoptotic effectors are largely unknown. Our understanding of the pathways of mitochondrial fission
, as often shown in textbooks, and a reticulum in which the organelle is a single, many-branched structure. The cell cycle– and metabolic state–dependent changes in mitochondrial morphology are controlled by a set of proteins that cause fission and fusion
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