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- 文献和实验
- 技术资料
- 保存条件:
"-20°C/-80°C"
- 保质期:
Generally, the shelf life of liquid form is 6 months at -20°C/-80°C. The shelf life of lyophilized form is 12 months at -20°C/-80°C.
- 英文名:
Customized Xenopus tropicalis tcta Protein (in vitro E.coli)
- 库存:
200
- 供应商:
武汉华美生物工程有限公司
- 规格:
20ug
Alternative Name(s)
T-cell leukemia translocation-altered gene protein homologEditorial/Sponsord
EditorialUniprot ID
A4IHD1Gene Names
tctaOrganism
Xenopus tropicalisAASequence
MAETWGSEIMTQALGCLQAFSSEFALEWENSDMKAAIFKLLLGWIVLSLTAIHLAWKSYG PTVNSIYYRQGMGGQNGGTPEYPARFPIWESSSTESLKRHQEExpression Region
1-102aaSequence Info
Full lengthSource
in vitro E.coliSource Notice
Mammalian cell expression systems and other species are available. Please inquire.Tag Info
InquireMW
InquireList Price
1384Purity
Greater than 85% as determined by SDS-PAGE.Storage Buffer
Tris/PBS-based buffer, 6% TrehaloseStorage
The shelf life is related to many factors, storage state, buffer ingredients, storage temperature and the stability of the protein itself. Generally, the shelf life of liquid form is 6 months at -20°C/-80°C. The shelf life of lyophilized form is 12 months at -20°C/-80°C.Endotoxin
Not Test. Endotoxin removal service is available for free upon you request.产品类型
Transmembrane-Protein备注
**产品信息可能有变动,请以官网信息为准
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文献和实验**产品信息可能有变动,请以官网信息为准
Spectral Karyotyping (SKY) of Hematologic Malignancies
The discovery of the Philadelphia chromosome in chronic myeloid leukemia (CML) by Novell and Hungerford in 1960 (1 ), the subsequent clarification of this chromosomal abnormality as areciprocal translocation t(9;22)(q34;q1 1) by Rowley
Analysis of Chromosomal Translocations
leukemia (CML) and a proportion of patients with acute lymphoblastic leukemia (ALL), the t(9;22)(q34;q11) translocation leads to the fusion of the ABL gene with part of the BCR gene. Locating the primers such that the polymerase chain reaction (PCR) product
CML Mouse Model in Translational Research
(1) with the Philadelphia chromosome resulting from of a reciprocal translocation between the chromosomes 9 and 22 t(9;22)-(q34;q11). This translocation produces a fusion gene known as BCR-ABL which acquires uncontrolled tyrosine kinase activity, constantly turning
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