GEMIN2抗体GEMIN2一抗抗体Survival of
motor neuron protein-interacting protein 1 antibody抗体GEMIN2 Antibody抗体- ￥1100 - 1980
- CUSABIO
- CN
- CSB-PA293526
- 2025年07月12日
- ELISA,IHC;ELISA:1:1000-1:5000,IHC:1:25-1:100
- Rabbit
- Human,Mouse,Rat
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- 详细信息
- 文献和实验
- 技术资料
- 免疫原：
Synthetic peptide of Human GEMIN2
- 亚型：
IgG
- 形态：
Liquid
- 保存条件：
Upon receipt, store at -20℃ or -80℃. Avoid repeated freeze.
- 克隆性：
/
- 标记物：
Non-conjugated
- 适应物种：
Human,Mouse,Rat
- 保质期：
6个月
- 抗原来源：
Homo sapiens (Human)
- 目录编号：
O14893
- 级别：
优
- 库存：
200
- 供应商：
武汉华美生物工程有限公司
- 宿主：
Rabbit
- 应用范围：
ELISA,IHC;ELISA:1:1000-1:5000,IHC:1:25-1:100
- 浓度：
>95%,Antigen affinity purification
- 靶点：
GEMIN2
- 抗体英文名：
GEMIN2 Antibody
- 抗体名：
Survival of motor neuron protein-interacting protein 1 antibody
- 规格：
100μl/50μl
| 规格： | 100μl | 产品价格： | ￥1980.0 |
|---|---|---|---|
| 规格： | 50μl | 产品价格： | ￥1100.0 |
保存缓冲液
-20℃, pH7.4 PBS, 0.05% NaN3, 40% Glycerol功能
This gene encodes one of the proteins found in the SMN complex, which consists of several gemin proteins and the protein known as the survival of motor neuron protein. The SMN complex is localized to a subnuclear compartment called gems (gemini of coiled bodies) and is required for assembly of spliceosomal snRNPs and for pre-mRNA splicing. This protein interacts directly with the survival of motor neuron protein and it is required for formation of the SMN complex. A knockout mouse targeting the mouse homolog of this gene exhibited disrupted snRNP assembly and motor neuron degeneration.风险提示：丁香通仅作为第三方平台，为商家信息发布提供平台空间。用户咨询产品时请注意保护个人信息及财产安全，合理判断，谨慎选购商品，商家和用户对交易行为负责。对于医疗器械类产品，请先查证核实企业经营资质和医疗器械产品注册证情况。
文献和实验：Transmembrane Protein GDE2 Induces Motor Neuron Differentiation in Vivo, Science , Vol 309, Issue 5744, 2212-2215 , 30 September 2005
Establishing In Vitro Models to Study Endogenous Neurotoxicants
aggregates in the trinucleotide repeat diseases (spinal bulbar muscular atrophy, Huntington’s disease, spinocerebellar ataxia 1 and 3, dentato-pallidoluysian atrophy) and cytoplasmic inclusions in familial and sporadic motor neuron diseases (45 ,46
. Keller‐Peck, C.R., Walsh, M.K., Gan, W.B., Feng, G., Sanes, J.R., and Lichtman, J.W. 2001. Asynchronous synapse elimination in neonatal motor units: Studies using GFP transgenic mice. Neuron 31:381‐394
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