- ￥776.90 - 1813.90
- 阿拉丁
- 上海
- F478729
- 2025年12月31日
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- 详细信息
- 文献和实验
- 技术资料
- 保存条件：
-20°C储存
- 英文名：
Fmoc-Phe(CF2PO3)-OH
- 库存：
现货
- 供应商：
上海阿拉丁生化科技股份有限公司
- 规格：
F478729-50mg/F478729-25mg/F478729-100mg
| 规格： | F478729-50mg | 产品价格： | ￥1166.9 |
|---|---|---|---|
| 规格： | F478729-25mg | 产品价格： | ￥776.9 |
| 规格： | F478729-100mg | 产品价格： | ￥1813.9 |
英文名：Fmoc-Phe(CF2PO3)-OH
纯度或规格：≥99%
SPU：F478729
CAS：160751-44-0
存储温度：-20°C储存
运输条件：超低温运输
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文献和实验Development of Conditionally Immortalized Epithelial Cell Lines from CF and Non-CF Mice
Mutations in the cystic fibrosis transmembrane conductance regulator (CFTR) gene are responsible for cystic fibrosis (CF) (1 ) and the primary defect in CF is the loss of cAMP-regulated anion conductance in the apical plasma membrane
Inflammatory Mediators in CF Patients
Patients with cystic fibrosis (CF) succumb to airway infection and inflammation, and determining the extent and nature of the infecting agents, as well as the extent and nature of the inflammatory response, is critical to understanding
Xenograft Model of the CF Airway
in associating cystic fibrosis (CF) defects with CFTR dysfunction, there are many unanswered questions concerning the roles of CFTR in both normal airway biology and in CF pathology. Before effective therapeutic approaches for CF lung disease will be realized
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