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- 2025年12月05日
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产品活性：BCR-ABL kinase-IN-3 (example 1) 是 BCR-ABL 的有效抑制剂，在急性髓性白血病 (AML) 研究中发挥着重要作用。
产品来源： www.medchemexpress.cn/bcr-abl-kinase-in-3.html
研究领域：Protein Tyrosine Kinase/RTK
作用靶点：Bcr-Abl
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文献和实验Fluorescence In Situ Hybridization for BCR-ABL
leukemia (CML) (1 ). A variable sequence length of the BCR gene at 22q11 fuses with ABL at 9q34 and encodes the constitutively active BCR-ABL protein tyrosine kinase (reviewed in refs. 2 and 3 ). Data from animal models have demonstrated
The use of oligodeoxynucleotides (ODN) to disrupt gene function has been studied in a variety of in vitro and in vivo systems (1 –14 ). Antigene, antisense, ribozyme, and aptamer nucleic acid molecules have been shown in numerous model
Detection of BCR-ABL Mutations and Resistance to Imatinib Mesylate
The major mechanism of imatinib resistance for patients with chronic myeloid leukemia (CML) is clonal expansion of leukemic cells with mutations in the Bcr-Abl fusion tyrosine kinase that reduce the capacity of imatinib to inhibit kinase
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