SNRPN Recombinant Mouse mAb(bsm-60533M)-50ul/100ul/25ul

SNRPN Recombinant Mouse mAb(bs

m-60533M)-50ul/100ul/25ul
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  • ¥800 - 2500
  • Bioss已认证
  • bsm-60533M
  • 2025年10月24日
  • 产品信息以Bioss网站为准
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    • 规格

      50ul/100ul/25ul

    规格:50ul产品价格:¥1400.0
    规格:100ul产品价格:¥2500.0
    规格:25ul产品价格:¥800.0
    产品编号bsm-60533M
    英文名称SNRPN Recombinant Mouse mAb
    产品应用WB=1:500-1000

    Not yet tested in other applications.
    Optimal working dilutions must be determined by the end user.

    交叉反应 (Human, Mouse, Rat)
    抗体来源Mouse
    亚型IgG1, Kappa
    性状Liquid
    纯化方法affinity purified by Protein G
    克隆类型Recombinant
    浓度1mg/ml
    储存液0.01M TBS (pH7.4) with 1% BSA, 0.02% Proclin300 and 50% Glycerol.
    研究领域

    Epigenetics and Nuclear Signaling > DNA / RNA > RNA Processing

    Epigenetics and Nuclear Signaling > DNA / RNA > Translation > Ribosome

    亚细胞定位Nucleus.
    组织特异性Expressed in brain and lymphoblasts.
    相似性Belongs to the snRNP SmB/SmN family.
    功能May be involved in tissue-specific alternative RNA processing events.
    保存条件Shipped at 4℃. Store at -20℃ for one year. Avoid repeated freeze/thaw cycles.
    注意事项This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications.
    背景资料This gene is located within the Prader-Willi Syndrome critical region on chromosome 15 and is imprinted and expressed from the paternal allele. It encodes a component of the small nuclear ribonucleoprotein complex, which functions in pre-mRNA processing and may contribute to tissue-specific alternative splicing. Alternative promoter use and alternative splicing result in a multitude of transcript variants encoding the same protein. Transcript variants that initiate at the CpG island-associated imprinting center may be bicistronic and also encode the SNRPN upstream reading frame protein (SNURF) from an upstream open reading frame. In addition, long spliced transcripts for small nucleolar RNA host gene 14 (SNHG14) may originate from the promoters at this locus and share exons with this gene. Alterations in this region are associated with parental imprint switch failure, which may cause Angelman syndrome or Prader-Willi syndrome. [provided by RefSeq, Mar 2017]

     

    应用推荐稀释比例
    {WB}{1:500-1000}

     

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