Phospho-Glucocorticoid Recepto
r (Ser211) Rabbit Polyclonal Antibody (PerCP) 抗体，orb1581699，Biorbyt- ￥6201
- Biorbyt
- orb1581699
- 英国
- 2026年08月06日
- FC
- Rabbit
- Human, Mouse, Rabbit
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- 文献和实验
- 技术资料
- 抗体名：
Phospho-Glucocorticoid Receptor (Ser211) Rabbit Polyclonal Antibody (PerCP) 抗体
- 抗体英文名：
Phospho-Glucocorticoid Receptor (Ser211) Rabbit Polyclonal Antibody (PerCP)
- 靶点：
NR3C1
- 浓度：
1mg/ml
- 应用范围：
FC
- 宿主：
Rabbit
- 适应物种：
Human, Mouse, Rabbit
- 保质期：
12 months from date of receipt.
- 抗原来源：
详询
- 目录编号：
orb1581699
- 级别：
科研
- 库存：
99
- 供应商：
Biorbyt
- 标记物：
PerCP
- 克隆性：
Polyclonal
- 保存条件：
详询
- 亚型：
IgG
- 免疫原：
详询
- 规格：
100 ul
产品子类型: Primary Antibody
描述: Phospho-Glucocorticoid Receptor (Ser211) Rabbit Polyclonal Antibody (PerCP) is a PerCP conjugated antibody targeting NR3C1. This antibody is suitable for FC. It exhibits reactivity with Human, Mouse, Rabbit samples.
别名: Glucocorticoid Receptor beta(phospho S211); Glucocorticoid Receptor(phospho S211); p-Glucocorticoid Receptor(phospho S211); Glucocorticoid Receptor; GCCR; GCR; GR; Nuclear receptor subfamily 3 group C member 1; Glucocorticoid receptor lymphocyte; GRL; Grl1; Nr3c1; NR3C1; GCR_HUMAN.
应用稀释比例: Flow-Cyt=1ug/Test
免疫原: KLH conjugated Synthesised phosphopeptide derived from human Glucocorticoid Receptor around the phosphorylation site of Ser211 NE(p-S)PW
预测反应性: Bovine, Equine, Rat
防腐剂: 0.01M TBS (pH7.4) with 1% rAlbumin and 0.02% Proclin300.
纯化方法: Affinity purified by Protein A
研究领域: Signal Transduction
保存条件: Store at 2-8°C for one year. Avoid repeated freeze/thaw cycles.
备注: For research use only.
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文献和实验dongreyuan 有的抗体名称上面写着Ab-33，比如，β-catenin（Ab-33）antibody，请问，Ab-33是什么意思？ bettyzyj 这个抗体是非磷酸化的抗体non-phospho-antibody ，33 代表的是基因位点 dongreyuan 谢谢了 yhwangcams bettyzyj wrote
Preparation of Polyclonal Antibodies to Retinoid Receptors
of a retinoid “X” receptor α (RXRα) antibody on Western blots contributed to the identification of the RXR ligand by detection of the receptor in fractions of whole-cell lysates that were able to bind the 9-cis retinoic acid ligand (2 ) Antibodies
), and causes activation of KOR. Agonist-activated KOR becomes a substrate for G protein receptor kinase (GRK), which phosphorylates the Ser369 residue at the C-terminal tail of the receptor in the first step in the β-Arrestin-dependent desensitization cascade
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