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- ProSci
- 美国
- PSI-33-773-100ug
- 2025年07月15日
- IF, WB, IHC-P
- 小鼠
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- 详细信息
- 文献和实验
- 技术资料
- 免疫原：
Human recombinant protein was used as the immunogen for this CFTR antibody.
- 保存条件：
Aliquot and Store at 2-8℃
- 保质期：
1年
- 级别：
科研级别
- 库存：
100
- 供应商：
艾美捷科技
- 宿主：
小鼠
- 应用范围：
IF, WB, IHC-P
- 抗体英文名：
CFTR Antibody [CFTR/1341]
- 抗体名：
CFTR Antibody [CFTR/1341]
- 规格：
100ug
CFTR 抗体 [CFTR/1341]，CFTR Antibody [CFTR/1341]，CFTR 抗体 [CFTR/1341],CFTR Antibody [CFTR/1341]
产品名称：CFTR 抗体 [CFTR/1341]-CFTR Antibody [CFTR/1341]
产品货号：PSI-33-773-100ug
产品规格：100ug
背景资料：Cystic fibrosis transmembrane conductance regulator (CFTR) is composed of two membrane-spanning domains (MSD), two nucleotide-binding domains (NBD), and an R domain. It is structurally similar to multidrug resistance (MDR1) protein and both are members of the superfamily of ATP-binding cassette (ABC) transporters, also known as traffic ATPases, which are implicated in the movement of various substrates. The protein is a small conductance adenosine 3',5'-cyclic monophosphate (cAMP)-activated chloride ion channel found in the apical membranes of epithelia within the pancreas, airway, intestine, bile duct, sweat gland, and male genital ducts. CFTR is a valuable marker of human pancreatic duct cell development and differentiation.
保存建议：CFTR 抗体 [CFTR/1341]-CFTR Antibody [CFTR/1341]Aliquot and Store at 2-8℃
应用类型：IF, WB, IHC-P
点击：CFTR 抗体 [CFTR/1341]-CFTR Antibody [CFTR/1341]查看更详细产品说明，更多应用、储存、价格、货期等信息请垂询艾美捷科技有限公司。更多ProSci公司特色试剂盒、特色抗体以及特色试剂等产品评论，请点击查看艾美捷特色产品中心。
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文献和实验Cystic fibrosis (CF) is caused by mutations in the cystic fibrosis transmembrane conductance regulator (CFTR ) gene (1 ,2 ). More than 1000 disease mutations and 200 polymorphisms have been identified in the CFTR gene (3 ). The type
mutation db Antithrombin mutation db Asthma and Allergy gene db BIOMDB - Db of mutations causing tetrahydrobiopterin deficiencies BLMbase - Human BLM mutation db (Bloom snydrom) BTKbase - Human BTK mutation db (X-linked
遗传异质性是由于基因变异 ( 其中绝大多数为 SNP) 引起的 遗传 异质性是由于基因变异(其中绝大多数为SNP)引起的，如APOE*E4与老年性痴呆、V因子1691G+A与深静脉血栓形成存在很强的相关性。不同基因的突变与不同的表型相关，但事实上同一基因的突变可能与许多疾病表型相关。例如，ATRX转录因子的不同突变导致不同表型的智力低下。在其他一些情况下，单个基因的突变甚至可以产生毫不相关的多种异常表型。如果携带2个无效CFTR等位基因的患者患严重的囊性
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