- ￥7000
- GeneTex
- 美国
- GTX34924
- 2025年07月08日
- WB, ICC/IF, IHC-P
- Mouse
- Human
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- 详细信息
- 文献和实验
- 技术资料
- 免疫原：
Recombinant full-length human CDKN1A protein
- 亚型：
IgG2a
- 形态：
Liquid
- 保存条件：
Store as concentrated solution. Centrifuge briefly prior to opening vial. For short-term storage (1-2 weeks), store at 4ºC. For long-term storage, aliquot and store at -20ºC or below. Avoid multiple freeze-thaw cycles.
- 克隆性：
Monoclonal
- 标记物：
Unconjugated
- 适应物种：
Human
- 保质期：
12 months from the shipping date of the product.
- 抗原来源：
Human
- 目录编号：
GTX34924
- 级别：
Primary Antibodies
- 库存：
Available
- 供应商：
GeneTex
- 宿主：
Mouse
- 应用范围：
WB, ICC/IF, IHC-P
- 浓度：
0.2 mg/ml (Please refer to the vial label for the specific concentration.)
- 靶点：
This MAb recognizes a 21kDa protein, identified as the p21WAF1 tumor suppressor protein. This MAb is highly specific to p21 and shows no cross-reaction with other closely related mitotic inhibitors.,We do not recommend use of this product for Mouse,Rat.
- 抗体英文名：
p21 Cip1 antibody [CIP1/823]
- 抗体名：
p21 Cip1 抗体 [CIP1/823]
- 规格：
100 μg
IHC-P analysis of human bladder carcinoma tissue using GTX34924 p21 Cip1 antibody [CIP1/823].
ICC/IF analysis of HeLa cells using GTX34924 p21 Cip1 antibody [CIP1/823].
Red : Primary antibody
Green : Phalloidin
IHC-P analysis of human lung sqcc tissue using GTX34924 p21 Cip1 antibody [CIP1/823].
WB analysis of HeLa cell lysate using GTX34924 p21 Cip1 antibody [CIP1/823].
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文献和实验Analysis of p21CDKN1A Recruitment to DNA Excision Repair Foci in the UV-Induced DNA Damage Response
The cyclin-dependent kinase (CDK) inhibitor p21CDKN1A (also known as p21waf1/cip1 ) is a well known player of the G1 and G2 phase cell cycle checkpoints, which are activated in response to DNA damage. In addition, p21 interacts directly
E2F Transcription Factors and pRb Pocket Proteins in Cell Cycle Progression
association with retinoblastoma pocket protein (pRb) family members. In turn, pRb is regulated through phosphorylation by cyclin-dependent kinase (cdk). The activity of cdk is negatively regulated by cdk-inhibitors, exemplified by p16INK4a , p21CIP1 , and p27
-induced responses, including a down-regulation of the cyclin-dependent kinase (Cdk2) inhibitors p21CIP1 and p27KIP1 , while attachment to P-L-L did not. We conclude that engagement of the FN-receptor integrin α5β1 induces integrin signaling regulating
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