品牌商
Cayman Chemical Company，Inc.
入驻年限：4 年
- 联系人：
Kristina Whitfield
- 所在地区：
美国
- 业务范围：
试剂、抗体、细胞库 / 细胞培养、ELISA 试剂盒
- 经营模式：
生产厂商
推荐产品
技术资料/正文
Ferroptosis
118 人阅读发布时间：2022-04-11 13:29
Cayman Educational Wall Poster Collection
Cayman Chemical has developed a series of informative wall posters that illustrate various lipid signaling and metabolic pathways as well as cellular processes related to ferroptosis, neutrophil biology, lipids, and more. These posters are available as a free resource for non-commercial purposes. You may log in to download a PDF copy of this poster or request a physical copy.
Ferroptosis
Ferroptosis is a type of cell death driven by iron-dependent phospholipid peroxidation that has been associated with various pathophysiologies. It is regulated by multiple cellular metabolic pathways, including redox homeostasis, iron handling, mitochondrial activity, and metabolism of amino acids, lipids, and sugars. Ferroptosis-inducing factors can directly or indirectly affect glutathione peroxidase or the ferroptosis suppressor protein, resulting in a decrease in antioxidant capacity and accumulation of lipid reactive oxygen species (ROS) in cells. This poster summarizes the current understanding of this cell death process with a focus on the various metabolic processes involved.
Cayman Chemical has developed a series of informative wall posters that illustrate various lipid signaling and metabolic pathways as well as cellular processes related to ferroptosis, neutrophil biology, lipids, and more. These posters are available as a free resource for non-commercial purposes. You may log in to download a PDF copy of this poster or request a physical copy.
Ferroptosis
Ferroptosis is a type of cell death driven by iron-dependent phospholipid peroxidation that has been associated with various pathophysiologies. It is regulated by multiple cellular metabolic pathways, including redox homeostasis, iron handling, mitochondrial activity, and metabolism of amino acids, lipids, and sugars. Ferroptosis-inducing factors can directly or indirectly affect glutathione peroxidase or the ferroptosis suppressor protein, resulting in a decrease in antioxidant capacity and accumulation of lipid reactive oxygen species (ROS) in cells. This poster summarizes the current understanding of this cell death process with a focus on the various metabolic processes involved.
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